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Ventolin to Salamol—a crossover study in New
Zealand
Shane Reti
In July 1 2005, PHARMAC (the New Zealand body responsible
for government-funded pharmaceutical subsidies) removed the subsidy on the
Ventolin® metered dose inhaler (MDI) (salbutamol – GlaxoSmithKline) in
favour of a chlorofluorocarbon (CFC)-free equivalent, Salamol® MDI
(salbutamol – Baker Norton).
From February 2005, the government agency responsible for
monitoring adverse drug reactions—Centre for Adverse Reactions Monitoring
(CARM)—noted increasing reports relating to patients crossing over from
Ventolin to Salamol, even “exceeding the normal capacity of CARM’s
processing systems, and exceeding the usual reporting rate for brand switching
complaints”.1 The three main complaints
were decreased therapeutic effect, blockage, and taste. Paediatricians also
reported particular concerns for children converting to Salamol, and questioned
the overall cost effectiveness of the
crossover.2
A formal investigation was undertaken by Medsafe, the
government agency responsible for registering pharmaceuticals, which primarily
examining the functionality of both new (16 inhalers) and returned faulty
inhalers (33 inhalers) against such measures as dose deposition, content
uniformity, and average dose per actuation.
Medsafe’s report was published in December 2005 with
the main finding pointing to device blockage as the likely main cause for
decreased therapeutic effect.1 Increased
patient education and adherence to the manufacturer’s weekly cleaning
recommendations was the suggested solution. Under these conditions, the testing
of Salamol appeared to pass appropriate laboratory tests of functionality,
however it is a completely different issue to then discuss the clinical
implications for asthmatics and the overall effects on their asthma
stability”.
Several studies have demonstrated clinical equivalence
between various salbutamol-containing MDIs with and without CFCs.
3–8 One question is whether these
findings can be generalised to all CFC-free salbutamol-containing MDIs, or
whether company specific formulations and MDI design have differences that
matter for asthma stability. Lumry et al drew attention to CFC-free salbutamol
MDIs variably containing “excipients such as oleic acid, lecithin, or
alcohol”.9
Lee has suggested that patient familiarity with the physical
aspects of salbutamol inhalers is important for
asthmatics.10 To this effect, there are few
studies that specifically compare brand name Salamol with Ventolin as is being
introduced in New Zealand. A 1996 randomised crossover study of 10 patients
found both to be equally effective,11 as did a
1994 crossover study of 11 patients.12
Comparing Salamol and Ventolin in the current New Zealand
setting is significant in that New Zealand has the one of the highest asthma
rates in the world (15% of adults, 20% of
children13), the range of available asthma
inhalers is less than in larger countries, and medication choice is mostly
driven by government subsidies.
Ventolin has been the main stay of beta-agonist treatment
for many years. In this context, this study examines the effects of converting
asthma patients from Ventolin to Salamol.
MethodSubjects were computer selected from the general
practice of the author with the following criteria:
Subjects then met
with the author (SR) who explained that the study was exploring the conversion
from Ventolin to Salamol, that the author had faith in both, and safety measures
associated with the study. Signed patient consent to participate was then
obtained.
The author then applied the validated New Zealand
version of the Asthma Control Questionnaire which is a combination of validated
questions on asthma symptoms, limitations, inhaler usage, and a peak flow
measurement.14 Demographic questions on age,
gender and ethnicity were also asked.
Subjects were then given a prescription for Salamol,
and instructed to completely replace Ventolin with Salamol for a period of 4
weeks. All existing medications, asthma and non-asthma related, were to remain
unchanged.
Four weeks later, the patients were recalled for
follow-up and the Asthma Control Questionnaire reapplied. During the study, any
subjects who were unable to complete the study, or who had to return to
Ventolin, were returned for consultation, and appropriate questioning made with
responses recorded.
As per the questionnaire validation, a change in asthma
stability was accepted as being a change of > 0.5 points on the questionnaire
scale. The statistical means were calculated for all categories, with the
standard error of the mean reported as 95% confidence intervals.
ResultsThirty-six subjects were initially enrolled in the study, 21
women and 15 men with an age range of between 17–76, and an average age of
48.83 years. None had hospital admissions for asthma in the previous year; 18/36
(50%) were using a preventer daily, and the average percentage predicted peak
expiratory flow rate was 70.11%.
Of the 36, 6/36 (17%; 95%CI 4–29%) withdrew during the
month-long study period; 5 of the 6 (83%, 95% CI 53 – 100%) withdrew due
to Salamol ineffectiveness, and the remaining subject withdrew citing the
unpleasant taste of Salamol.
A further 15/36 of the original subjects (42%; 95%CI
25–58%) could not maintain Salamol alone, and had to return to Ventolin at
some point during the month.
Of the 15 who returned to Ventolin, 10/15 (67%, 95%CI
42–91%) cited Salamol ineffectiveness, 1/15 device blockage, 3/15 the
convenience of having Ventolin handy, and 1/15 gave no reason.
This left 15/36 (42%; 95%CI 25–58%) of the original
subjects successfully maintaining ‘Salamol only’ as per the study
design. Figure 1 shows a flow diagram of participant outcomes.
Assessment of asthma stability with the asthma control
questionnaire showed 14/15 (93%; 95%CI 80–100%) had worse asthma
stability, and the remaining subject had unchanged asthma stability; 2 of the 15
(13%; 95%CI 0–31%), who maintained Salamol only, gave possible influences
on their asthma over the study period—both citing chest infections.
Figure 1. Flow diagram of participant outcomes
![]() DiscussionOnly 15 subjects out of 36 in this study were able to
maintain the study design using Salamol; and in this group of 15, 93% has worse
asthma stability.
For all study participants, Salamol ineffectiveness was a
significant factor. However these findings need to be considered in the context
of several potential limitations. Firstly, study numbers are small (this
particularly limits demographic analysis), although consistent in size with
several other asthma inhaler crossover
studies.11,12,15
Secondly, there are external variables such as the weather,
pollen counts, winter chest infections, change in home or work environment or
lifestyle, change in preventer use, and negative media that could have had an
influence over the month-long study period. Most of these variables are
discussed under the heading “Peripheral Related Factors”, and to
summarise here, are not considered to be significant influences in this study.
Self directed changes in steroid usage could influence the
findings, however none of the subjects completing the study reported changed
their steroid inhaler usage. Furthermore, Bleecker et al demonstrated the
non-significance of inhaled steroid use in a similar crossover study comparing
Airomir® CFC-free (another CFC-free formulation of salbutamol) and
Ventolin.16
There are several explanations for the findings of apparent
ineffectiveness and deteriorated asthma stability on the crossover to Salamol.
These can be considered under the headings patient-related, peripheral-related,
or product-related factors.
Patient-related factors—Patient
related factors primarily relate to how change alone, and not Salamol
ineffectiveness, may contribute to the findings.
Indeed, anxiety, apprehension, and caution are likely to be
the norm in a crossover study of this type, possibly precipitating an early
return to a previous trusted inhaler. This uncertainty under change was
demonstrated in a crossover study of 29 patients using either branded
salbutamol, generic salbutamol, or their usual salbutamol (blinded).
Juniper concluded “patients’ own assessment of
their relief inhaler seems to be influenced by factors other than
efficacy”.15 If change is accepted as an
operative factor, then with these two products the effect was further magnified
by Salamol being significantly smaller in size than Ventolin, having a less
forceful particle actuation, and having a different taste.
Lee has suggested that patient familiarity with the physical
aspects of salbutamol inhalers is important for
asthmatics.10 While change-related patient
factors may well have contributed to subjects returning to Ventolin and
reporting ineffectiveness, these factors cannot fully account for the 93% who
maintained Salamol only, and who by ACQ testing had worse asthma stability.
Peripheral-related factors—These
factors are outside the control of the study design and are known to have an
influence in asthma, and could possibly have contributed to Salamol
ineffectiveness. These factors include weather, pollen, chest infections, change
in home or work environment or lifestyle, and negative media.
To assess the effect of these external factors, subjects
were asked if they were aware of anything that might have altered their asthma
over the study period. This form of subjective self-analysis is more blunt than
definitive, however it did contribute some useful information suggesting a small
influence particularly from chest infections.
External effects were also minimised by choosing a month
that was not a recognised change of season month, nor overtly mid-winter.
Salamol had also received mostly negative reporting in the
media up to 5 months prior to the study onset. The effect of this on this study
is difficult to assess, however it was mitigated as far as possible through
patient education in the consent process, and the unbiased support for Salamol
from the author as their family doctor. Overall, the influence of these
peripheral factors is considered to be minimal.
Product-related factors—The apparent
ineffectiveness of Salamol in this crossover study is in contrast to the few
previously mentioned crossover studies from Ventolin to Salamol.
In this study, subjects complained about the taste, device
blockage, and the uncertainty of whether they were getting anything in their
mouth after using Salamol.
Other studies with similar products e.g. Ventolin and
Aeromir (another CFC-free formulation of salbutamol) demonstrate a slower
particle speed for the CFC-free formulation that may account for the delivery
uncertainty.17
Bamber reported blockage difficulties with CFC-free inhalers
and Chew confirmed blockage-induced reductions in fine particle mass requiring
weekly mouthpiece washing.18,19 These
device-related factors may certainly account for the observed poor asthma
stability, and to this must be added the final possibility that the absolute
effectiveness of CFC-free salbutamol in Salamol is simply not as effective as
that in Ventolin.
It is possible that inter-company preferences for non-active
ingredients such as excipients eg alcohol, alters the effectiveness of
Salbutamol itself.
ConclusionsWhile promoted as being pharmaceutically similar, Salamol
was less effective than Ventolin in this study. This could be due to several
factors, including true differences in active ingredient efficacy, physical
differences in inhaler devices, and subject-related change anxiety. Further work
needs to be done to identify these individual factors with greater recognition
especially, of the “change-related” contributions to asthma.
If the physical delivery features of a device are different,
and patients are not adequately reassured, educated, and safely trialled, then
it is highly likely any new asthma inhaler introduction will face difficulties
no matter how bioequivalent it may pharmaceutically turn out to be.
Disclosures: The author of this article is
an independent researcher and has not been the recipient of any funding from
GlaxoSmithKline (the manufacturer of Ventolin), or funding from any other
source.
Author information: Shane Reti, Medical
Practitioner and Researcher, Whangarei (and Senior Lecturer, School of
Population Health, Auckland University, Auckland)
Acknowledgments: I thank Allen Liang for
peer review and Mike Mullany for statistical support.
Correspondence: Dr Shane Reti, 15 Rust Ave,
Whangarei. Fax (09) 438 2011; email: salamol@selectpost.com
References:
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