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A 14-year-old girl presented with gradually increasing abdominal distension for 6 months. She recently noticed mild breathlessness on exertion. Examination revealed a tense ascites with mild oedema of the lower limbs. Her jugular venous pulse (JVP) was not seen to be elevated in the supine or 45 00ba position but was raised up to the ear lobes in sitting up position. A pericardial knock was heard on auscultation.Lateral X-ray of the chest showed calcification of the pericardium (Figure 1). CT scan showed a circumferential calcification of the pericardium (Figure 2) along with massive ascites (Figure 3). Figure 1. X-ray chest (lateral view) Figure 2. CT scan of the chest Figure 3. CT scan of the abdomen Echocardiography findings were consistent with constrictive pericarditis. She underwent a pericardiectomy with complete resolution of symptoms. Tuberculosis was confirmed as the cause of chronic calcific constrictive pericarditis on the histologic sections of the pericardium postoperatively. Ascites secondary to constrictive pericarditis typically occurs before the oedema of the lower limbs, unlike other cardiac causes. Hence it is referred to as ascites praecox. Abdominal signs such as hepatomegaly and ascites frequently over shadow the cardiac signs causing difficulty in diagnoses. The most important physical sign in constrictive pericarditis is a raised JVP which is often missed as it is grossly elevated above the angle of the jaw. A sitting up position is preferred in such cases. The importance of making an accurate diagnosis lies in the fact that surgical intervention can provide complete relief of symptoms in these patients.

Summary

Abstract

Aim

Method

Results

Conclusion

Author Information

Pazhanivel Mohan, Senior Registrar; Jayanthi Venkataraman, Professor of Gastroenterology Department of Gastroenterology, Stanley Medical College, Royapuram, Chennai, India

Acknowledgements

Correspondence

Pazhanivel Mohan, MD, Department of Gastroenterology, Stanley Medical College, Old jail road, Royapuram, Chennai - 600001, India.

Correspondence Email

dr.pazhani@gmail.com

Competing Interests

For the PDF of this article,
contact nzmj@nzma.org.nz

View Article PDF

A 14-year-old girl presented with gradually increasing abdominal distension for 6 months. She recently noticed mild breathlessness on exertion. Examination revealed a tense ascites with mild oedema of the lower limbs. Her jugular venous pulse (JVP) was not seen to be elevated in the supine or 45 00ba position but was raised up to the ear lobes in sitting up position. A pericardial knock was heard on auscultation.Lateral X-ray of the chest showed calcification of the pericardium (Figure 1). CT scan showed a circumferential calcification of the pericardium (Figure 2) along with massive ascites (Figure 3). Figure 1. X-ray chest (lateral view) Figure 2. CT scan of the chest Figure 3. CT scan of the abdomen Echocardiography findings were consistent with constrictive pericarditis. She underwent a pericardiectomy with complete resolution of symptoms. Tuberculosis was confirmed as the cause of chronic calcific constrictive pericarditis on the histologic sections of the pericardium postoperatively. Ascites secondary to constrictive pericarditis typically occurs before the oedema of the lower limbs, unlike other cardiac causes. Hence it is referred to as ascites praecox. Abdominal signs such as hepatomegaly and ascites frequently over shadow the cardiac signs causing difficulty in diagnoses. The most important physical sign in constrictive pericarditis is a raised JVP which is often missed as it is grossly elevated above the angle of the jaw. A sitting up position is preferred in such cases. The importance of making an accurate diagnosis lies in the fact that surgical intervention can provide complete relief of symptoms in these patients.

Summary

Abstract

Aim

Method

Results

Conclusion

Author Information

Pazhanivel Mohan, Senior Registrar; Jayanthi Venkataraman, Professor of Gastroenterology Department of Gastroenterology, Stanley Medical College, Royapuram, Chennai, India

Acknowledgements

Correspondence

Pazhanivel Mohan, MD, Department of Gastroenterology, Stanley Medical College, Old jail road, Royapuram, Chennai - 600001, India.

Correspondence Email

dr.pazhani@gmail.com

Competing Interests

For the PDF of this article,
contact nzmj@nzma.org.nz

View Article PDF

A 14-year-old girl presented with gradually increasing abdominal distension for 6 months. She recently noticed mild breathlessness on exertion. Examination revealed a tense ascites with mild oedema of the lower limbs. Her jugular venous pulse (JVP) was not seen to be elevated in the supine or 45 00ba position but was raised up to the ear lobes in sitting up position. A pericardial knock was heard on auscultation.Lateral X-ray of the chest showed calcification of the pericardium (Figure 1). CT scan showed a circumferential calcification of the pericardium (Figure 2) along with massive ascites (Figure 3). Figure 1. X-ray chest (lateral view) Figure 2. CT scan of the chest Figure 3. CT scan of the abdomen Echocardiography findings were consistent with constrictive pericarditis. She underwent a pericardiectomy with complete resolution of symptoms. Tuberculosis was confirmed as the cause of chronic calcific constrictive pericarditis on the histologic sections of the pericardium postoperatively. Ascites secondary to constrictive pericarditis typically occurs before the oedema of the lower limbs, unlike other cardiac causes. Hence it is referred to as ascites praecox. Abdominal signs such as hepatomegaly and ascites frequently over shadow the cardiac signs causing difficulty in diagnoses. The most important physical sign in constrictive pericarditis is a raised JVP which is often missed as it is grossly elevated above the angle of the jaw. A sitting up position is preferred in such cases. The importance of making an accurate diagnosis lies in the fact that surgical intervention can provide complete relief of symptoms in these patients.

Summary

Abstract

Aim

Method

Results

Conclusion

Author Information

Pazhanivel Mohan, Senior Registrar; Jayanthi Venkataraman, Professor of Gastroenterology Department of Gastroenterology, Stanley Medical College, Royapuram, Chennai, India

Acknowledgements

Correspondence

Pazhanivel Mohan, MD, Department of Gastroenterology, Stanley Medical College, Old jail road, Royapuram, Chennai - 600001, India.

Correspondence Email

dr.pazhani@gmail.com

Competing Interests

Contact diana@nzma.org.nz
for the PDF of this article

View Article PDF

A 14-year-old girl presented with gradually increasing abdominal distension for 6 months. She recently noticed mild breathlessness on exertion. Examination revealed a tense ascites with mild oedema of the lower limbs. Her jugular venous pulse (JVP) was not seen to be elevated in the supine or 45 00ba position but was raised up to the ear lobes in sitting up position. A pericardial knock was heard on auscultation.Lateral X-ray of the chest showed calcification of the pericardium (Figure 1). CT scan showed a circumferential calcification of the pericardium (Figure 2) along with massive ascites (Figure 3). Figure 1. X-ray chest (lateral view) Figure 2. CT scan of the chest Figure 3. CT scan of the abdomen Echocardiography findings were consistent with constrictive pericarditis. She underwent a pericardiectomy with complete resolution of symptoms. Tuberculosis was confirmed as the cause of chronic calcific constrictive pericarditis on the histologic sections of the pericardium postoperatively. Ascites secondary to constrictive pericarditis typically occurs before the oedema of the lower limbs, unlike other cardiac causes. Hence it is referred to as ascites praecox. Abdominal signs such as hepatomegaly and ascites frequently over shadow the cardiac signs causing difficulty in diagnoses. The most important physical sign in constrictive pericarditis is a raised JVP which is often missed as it is grossly elevated above the angle of the jaw. A sitting up position is preferred in such cases. The importance of making an accurate diagnosis lies in the fact that surgical intervention can provide complete relief of symptoms in these patients.

Summary

Abstract

Aim

Method

Results

Conclusion

Author Information

Pazhanivel Mohan, Senior Registrar; Jayanthi Venkataraman, Professor of Gastroenterology Department of Gastroenterology, Stanley Medical College, Royapuram, Chennai, India

Acknowledgements

Correspondence

Pazhanivel Mohan, MD, Department of Gastroenterology, Stanley Medical College, Old jail road, Royapuram, Chennai - 600001, India.

Correspondence Email

dr.pazhani@gmail.com

Competing Interests

Contact diana@nzma.org.nz
for the PDF of this article

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